Summary Escalating evidence implicates altered mTORC1 signaling cascades within the pathophysiology of depression, suggesting that immediate modulation of mTORC1 signaling may well supply novel therapeutic opportunity. In this issue of the JCI, Kato and colleagues noted that administration of NV-5138, a not long ago made artificial leucine analog, incorporates a https://trichostatin-a24680.blog4youth.com/29954811/the-basic-principles-of-shield-1